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dc.rights.licenseopenen_US
hal.structure.identifierImmunology from Concept and Experiments to Translation [ImmunoConcept]
dc.contributor.authorSANTA, Pauline
hal.structure.identifierImmunology from Concept and Experiments to Translation [ImmunoConcept]
dc.contributor.authorGARREAU, Anne
dc.contributor.authorSERPAS, L.
hal.structure.identifierImmunology from Concept and Experiments to Translation [ImmunoConcept]
dc.contributor.authorFERRIERE, Amandine
hal.structure.identifierImmunology from Concept and Experiments to Translation [ImmunoConcept]
dc.contributor.authorBLANCO, Patrick
dc.contributor.authorSONI, C.
hal.structure.identifierImmunology from Concept and Experiments to Translation [ImmunoConcept]
dc.contributor.authorSISIRAK, Vanja
dc.date.accessioned2022-05-09T09:20:13Z
dc.date.available2022-05-09T09:20:13Z
dc.date.issued2021-04-22
dc.identifier.issn1664-3224en_US
dc.identifier.urihttps://oskar-bordeaux.fr/handle/20.500.12278/139999
dc.description.abstractEnIn the original article, there was a mistake in the legend for Figure 2 as published. The correct legend for Figure 2 was mistakenly omitted and replaced with the legend of figure 3. The correct legend appears below. DNASE1L3 deficiency leads to the accumulation of numerous forms of DNA including chromatin, MP associated DNA and NET-associated DNA. Accumulation of such DNA contributes to the aberrant activation of TLR7,9 in B cells and plasmacytoid dendritic cells (pDCs). In B cells TLR7,9 activation leads to their differentiation into plasma cells and antibody forming cells (AFC) that produce autoreactive antibodies mostly directed against dsDNA. In pDCs TLR7,9 activation induces the production of type I interferons (IFN-I) which also play an important role in the transition of B cells into AFC. The production of anti-dsDNA antibodies and of IFN-I will ultimately cause the development of Systemic Lupus Erythematosus (SLE). The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated. (Figure presented.).
dc.language.isoENen_US
dc.rightsAttribution 3.0 United States*
dc.rights.urihttp://creativecommons.org/licenses/by/3.0/us/*
dc.subject.enaicardi goutieres syndrome
dc.subject.enDNA sensing
dc.subject.enDNases
dc.subject.eninterferonopathies
dc.subject.enRNA sensing
dc.subject.enRNases
dc.subject.ensystemic lupus erythematosus
dc.subject.entoll-like receptors
dc.title.enCorrigendum: The Role of Nucleases and Nucleic Acid Editing Enzymes in the Regulation of Self-Nucleic Acid Sensing (Frontiers in Immunology, (2021), 12, (629922), 10.3389/fimmu.2021.629922)
dc.typeArticle de revueen_US
dc.identifier.doi10.3389/fimmu.2021.690853en_US
dc.subject.halSciences du Vivant [q-bio]/Immunologieen_US
dc.identifier.pubmed33968091en_US
bordeaux.journalFrontiers in Immunologyen_US
bordeaux.volume12en_US
bordeaux.hal.laboratoriesImmunoConcEpT - UMR 5164en_US
bordeaux.institutionUniversité de Bordeauxen_US
bordeaux.institutionCNRSen_US
bordeaux.peerReviewedouien_US
bordeaux.inpressnonen_US
hal.identifierhal-03662261
hal.version1
hal.date.transferred2022-05-09T09:20:16Z
hal.exporttrue
dc.rights.ccCC BYen_US
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